APTT sawv cev rau lub sijhawm ua haujlwm ib nrab thromboplastin, uas yog hais txog lub sijhawm yuav tsum tau ntxiv ib nrab thromboplastin rau cov ntshav kuaj ntshav thiab saib xyuas lub sijhawm xav tau rau cov ntshav coagulation.APTT yog ib qho rhiab heev thiab feem ntau siv tshuaj ntsuam xyuas rau kev txiav txim siab endogenous coagulation system.Qhov ib txwm muaj yog 31-43 vib nas this, thiab 10 vib nas this ntau tshaj li qhov kev tswj hwm ib txwm muaj qhov tseem ceeb hauv kev kho mob.Vim tias qhov sib txawv ntawm cov tib neeg, yog tias qib ntawm APTT luv luv me ntsis, nws kuj yuav yog qhov tshwm sim ib txwm muaj, thiab tsis tas yuav muaj kev ntshai heev, thiab kev kuaj xyuas tsis tu ncua yog txaus.Yog tias koj tsis xis nyob, mus ntsib kws kho mob raws sijhawm.

APTT shortening qhia tias cov ntshav nyob rau hauv lub xeev hypercoagulable, uas yog ib qho tshwm sim ntawm cov kab mob plawv thiab cerebrovascular thrombotic, xws li cerebral thrombosis thiab kab mob plawv.

1. Cerebral thrombosis

Cov neeg mob uas luv luv APTT feem ntau yuav tsim cov hlab ntsha hlwb, uas yog ib qho ntawm cov kab mob uas muaj feem xyuam rau cov ntshav hypercoagulation los ntawm kev hloov pauv hauv cov ntshav, xws li hyperlipidemia.Nyob rau lub sijhawm no, yog tias qib ntawm cerebral thrombosis yog qhov mob me, tsuas yog cov tsos mob ntawm cov ntshav tsis txaus rau lub hlwb yuav tshwm sim, xws li kiv taub hau, mob taub hau, xeev siab, thiab ntuav.Yog hais tias qhov degree ntawm cerebral thrombosis hnyav txaus ua rau mob cerebral parenchymal ischemia, cov tsos mob xws li kev ua haujlwm tsis zoo ntawm lub cev, kev hais lus tsis zoo, thiab kev tsis pom kev yuav tshwm sim.Rau cov neeg mob uas mob cerebral thrombosis, feem ntau yog siv cov pa oxygen nqus pa thiab kev ua pa tawm.Thaum tus neeg mob cov tsos mob ua rau muaj kev phom sij rau lub neej, kev ua haujlwm thrombolysis lossis kev phais yuav tsum tau ua kom qhib cov hlab ntsha sai li sai tau.Tom qab cov tsos mob tseem ceeb ntawm cerebral thrombosis tau txo qis thiab tswj tau, tus neeg mob tseem yuav tsum ua raws li kev coj noj coj ua zoo thiab noj tshuaj mus ntev raws li cov kws kho mob qhia.Nws raug nquahu kom noj cov zaub mov uas tsis muaj ntsev thiab cov rog tsawg thaum lub sijhawm rov qab los, noj zaub thiab txiv hmab txiv ntoo ntau dua, tsis txhob noj cov zaub mov uas muaj sodium xws li nqaij npuas kib, pickles, cov kaus poom, thiab lwm yam, thiab tsis txhob haus luam yeeb thiab cawv.Kev tawm dag zog me ntsis thaum koj tus mob lub cev tso cai.

2. Kab mob plawv

Qhov luv luv ntawm APTT qhia tau hais tias tus neeg mob yuav raug kev txom nyem los ntawm kab mob plawv, uas feem ntau tshwm sim los ntawm cov ntshav coronary hypercoagulation ua rau stenosis lossis blockage ntawm cov hlab ntsha lumen, ua rau muaj kev cuam tshuam ntawm myocardial ischemia, hypoxia, thiab necrosis.Yog tias qib ntawm cov hlab ntsha ntawm cov hlab ntsha yog qhov siab, tus neeg mob yuav tsis muaj cov tsos mob tshwm sim hauv lub xeev so, lossis tsuas yog muaj kev tsis xis nyob xws li hauv siab nruj thiab mob hauv siab tom qab ua haujlwm.Yog tias qib ntawm cov hlab ntsha ntawm cov hlab ntsha txhaws hnyav heev, qhov kev pheej hmoo ntawm myocardial infarction nce.Cov neeg mob yuav hnov ​​​​mob hauv siab, mob hauv siab, thiab ua pa luv thaum lawv so lossis kev xav zoo siab.Qhov mob tuaj yeem kis mus rau lwm qhov ntawm lub cev thiab nyob twj ywm yam tsis muaj kev pab.Rau cov neeg mob uas muaj mob plawv mob plawv, tom qab kev tswj hwm sublingual ntawm nitroglycerin lossis isosorbide dinitrate, mus ntsib kws kho mob tam sim ntawd, thiab tus kws kho mob soj ntsuam seb puas yuav tsum tau siv cov stent implantation los yog thrombolysis tam sim ntawd.Tom qab lub sijhawm mob hnyav, yuav tsum tau siv cov tshuaj antiplatelet ntev thiab anticoagulant.Tom qab tso tawm hauv tsev kho mob, tus neeg mob yuav tsum noj zaub mov tsis muaj ntsev thiab tsis muaj rog, txiav luam yeeb thiab haus dej haus cawv, tawm dag zog kom zoo, thiab ua tib zoo so.