Kev kho mob ntawm thrombosis feem ntau yog siv cov tshuaj tiv thaiv thrombotic, uas tuaj yeem qhib cov ntshav thiab tshem tawm cov ntshav stasis.Tom qab kev kho mob, cov neeg mob thrombosis xav tau kev cob qhia kev kho mob.Feem ntau, lawv yuav tsum ntxiv dag zog rau kev cob qhia ua ntej lawv tuaj yeem maj mam rov zoo.Kev pw tsaug zog ntev ntev tuaj yeem yooj yim ua rau muaj teeb meem ntawm thrombosis.Nws yog ib qho tsim nyog los ntxiv dag zog rau kev tawm dag zog tom qab kev kho mob vim tsis muaj peev xwm saib xyuas tus kheej hauv lub neej, bedridden.

Hais txog kev kho mob, tam sim no muaj peb txoj hauv kev tseem ceeb.

1. Kev kho mob thrombolytic.Nyob rau hauv thawj theem ntawm thrombus, thrombus nyob rau hauv cov hlab ntsha tseem yog ib tug tshiab thrombus.Yog tias cov thrombus tuaj yeem yaj thiab cov ntshav rov ua dua tuaj yeem ua tau, nws yuav yog qhov kev ntsuas tseem ceeb txhawm rau txhim kho kev ncig, tiv thaiv cov hlwb thiab txhawb kev ua haujlwm zoo.Yog tias tsis muaj contraindication rau kev kho thrombolytic, qhov ua ntej ntawm daim ntawv thov, qhov txiaj ntsig zoo dua.

2, anticoagulation therapy, txawm hais tias feem ntau cov kev tshawb fawb tau pom tias heparin anticoagulation txoj kev kho tsis zoo rau cov txiaj ntsig ntawm ischemia, tab sis tam sim no kev loj hlob infarction yog qhov qhia txog kev kho mob thaum muaj xwm txheej ceev, uas tau txais los ntawm cov kws tshawb fawb feem ntau.Yog tias cov xwm txheej uas ua rau muaj kev loj hlob tau txiav txim siab tias yuav ua rau mob loj heev thiab tsis muaj kev cuam tshuam txog kev ua haujlwm tsis zoo, kev kho heparin tseem yog thawj qhov kev xaiv, thiab cov kev kho mob feem ntau yog tso dej los yog txhaj tshuaj subcutaneous ntawm heparin.

3. Volume expansion dilution therapy, nthuav cov ntshav ntim yuav tsum tau nqa tawm thaum tus neeg mob tsis muaj teeb meem cerebral edema lossis mob plawv tsis txaus.