1. Kev ua neej nyob

Kev noj zaub mov (xws li tsiaj lub siab), haus luam yeeb, haus, thiab lwm yam yuav cuam tshuam rau kev kuaj pom;

2. Yam tshuaj

(1) Warfarin: feem ntau cuam tshuam rau PT thiab INR qhov tseem ceeb;
(2) Heparin: Nws feem ntau cuam tshuam rau APTT, uas tuaj yeem ncua ntev li 1.5 txog 2.5 zaug (hauv cov neeg mob kho nrog cov tshuaj tiv thaiv coagulant, sim sau cov ntshav tom qab cov tshuaj concentration txo qis lossis cov tshuaj tau dhau los ntawm nws ib nrab-lub neej);
(3) Tshuaj tua kab mob: Kev siv tshuaj tua kab mob loj tuaj yeem ua rau lub sijhawm ntev ntawm PT thiab APTT.Nws tau raug tshaj tawm tias thaum cov ntsiab lus ntawm penicillin nce mus txog 20,000 u / ML cov ntshav, PT thiab APTT tuaj yeem ua rau ntev dua 1 zaug, thiab tus nqi INR kuj tseem tuaj yeem ua rau ntev dua 1 zaug (Cov kab mob txawv txav ntawm coagulation induced los ntawm cov hlab ntsha. nodooperazone-sulbactam tau tshaj tawm)
(4) thrombolytic tshuaj;
(5) Cov tshuaj muaj roj emulsion tuaj yeem cuam tshuam nrog cov txiaj ntsig ntawm kev sim, thiab kev siv cov centrifugation siab tuaj yeem siv los txo qhov cuam tshuam hauv cov qauv ntshav lipid hnyav;
(6) Cov tshuaj xws li tshuaj aspirin, dipyridamole thiab ticlopidine tuaj yeem tiv thaiv platelet aggregation;

3. Kev sau cov ntshav:

(1) Qhov piv ntawm sodium citrate anticoagulant rau ntshav feem ntau yog 1: 9, thiab nws yog tov zoo.Nws tau raug tshaj tawm nyob rau hauv cov ntaub ntawv hais tias qhov nce los yog txo ntawm anticoagulant concentration muaj qhov cuam tshuam rau kev tshawb pom ntawm coagulation muaj nuj nqi.Thaum cov ntshav ntim nce los ntawm 0.5 mL, lub sij hawm ua kom cov ntshav tuaj yeem luv;thaum cov ntshav ntim qis los ntawm 0.5 mL, lub sijhawm ua ntshav tuaj yeem ua kom ntev;
(2) Ntaus tus ntsia hlau rau ntawm lub taub hau kom tiv thaiv cov ntaub so ntswg puas thiab sib xyaw ntawm exogenous coagulation yam;
(3) Lub sijhawm ntawm lub cuff yuav tsum tsis pub tshaj 1 min.Yog tias lub cuff raug nias nruj dhau los yog lub sijhawm ntev dhau lawm, qhov tseem ceeb VIII thiab cov ntaub so ntswg plasmin qhov chaw activator (t-pA) yuav raug tso tawm vim ligation, thiab cov ntshav txhaj tshuaj yuav hnyav dhau.Nws kuj tseem yog kev tawg ntawm cov qe ntshav uas ua rau lub coagulation system.

4. Lub sij hawm thiab qhov kub thiab txias cuam tshuam ntawm qhov kev tso kawm:

(1) Coagulation yam Ⅷ thiab Ⅴ tsis ruaj khov.Raws li lub sij hawm cia nce, qhov chaw cia kub nce, thiab cov dej coagulation maj mam ploj mus.Yog li ntawd, cov ntshav coagulation yuav tsum raug xa mus kuaj tsis pub dhau 1 teev tom qab sau, thiab qhov kev ntsuam xyuas yuav tsum ua kom tiav hauv 2 teev kom tsis txhob ua rau PT., APTT prolongation.(2) Rau cov qauv uas tsis tuaj yeem kuaj tau raws sijhawm, cov ntshav ntshav yuav tsum tau muab cais thiab khaws cia hauv qab lub hau thiab tso rau hauv tub yees ntawm 2 ℃ ~ 8 ℃.

5. Cov qauv mob hnyav / hnyav hemolysis thiab lipidemia

Cov qauv hemolyzed muaj cov dej coagulation zoo ib yam li platelet factor III, uas tuaj yeem ua rau lub sijhawm TT, PT, thiab APTT ntawm hemolyzed plasma thiab txo cov ntsiab lus ntawm FIB.

6. Lwm tus

Hypothermia, acidosis, thiab hypocalcemia tuaj yeem ua rau thrombin thiab coagulation yam tsis zoo.