Thrombosis feem ntau muaj peb yam hauv qab no:

1. Mob plawv thiab hlab ntsha intima raug mob

Lub intima ntawm ib txwm lub plawv thiab cov hlab ntsha yog qhov qub thiab du, thiab cov hlwb endothelial tsis zoo tuaj yeem cuam tshuam platelet adhesion thiab anticoagulation.Thaum lub hauv paus membrane puas lawm, lub coagulation system yuav qhib tau nyob rau hauv ntau txoj kev.

Thawj qhov kev puas tsuaj intima tso cov ntaub so ntswg coagulation (coagulation factor III), uas ua rau lub cev coagulation extrinsic.
Qhov thib ob, tom qab lub intima puas lawm, lub endothelial hlwb raug degeneration, necrosis, thiab shedding, exposing lub collagen fibers nyob rau hauv lub endothelium, li no activating lub coagulation factor XII ntawm endogenous coagulation system thiab pib lub endogenous coagulation system.Nyob rau hauv tas li ntawd, lub puas intima ua ntxhib, uas yog conducive rau platelet deposition thiab adhesion.Tom qab cov platelets adhered rupture, ntau yam platelet yam raug tso tawm, thiab tag nrho cov txheej txheem coagulation yog qhib, ua rau cov ntshav coagulate thiab tsim thrombus.
Ntau yam ntawm lub cev, tshuaj lom neeg thiab tshuaj lom neeg tuaj yeem ua rau muaj kev puas tsuaj rau cov hlab plawv intima, xws li endocarditis hauv swine erysipelas, pulmonary vasculitis hauv bovine pneumonia, equine parasitic arteritis, rov txhaj tshuaj rau tib qho ntawm cov leeg, raug mob thiab puncture ntawm cov hlab ntsha phab ntsa. thaum phais.

2. Kev hloov ntawm cov ntshav ntws

Feem ntau yog hais txog cov ntshav khiav qeeb, tsim cov vortex thiab cov ntshav ntws tawm.
Raws li ib txwm muaj, cov ntshav khiav ceev ceev, thiab cov qe ntshav liab, platelets thiab lwm yam khoom xyaw yog concentrated nyob rau hauv nruab nrab ntawm cov hlab ntsha, uas yog hu ua axial flow;Thaum cov ntshav khiav qeeb qeeb, cov qe ntshav liab thiab cov platelets yuav ntws ze rau ntawm phab ntsa ntawm cov hlab ntsha, hu ua side flow, uas ua rau cov ntshav txhaws.txaus ntshai uas tshwm sim.
Cov ntshav khiav qeeb qeeb, thiab cov hlwb endothelial yog hypoxic hnyav, ua rau cov degeneration thiab necrosis ntawm endothelial hlwb, poob ntawm lawv cov hauj lwm ntawm synthesizing thiab tso tawm anticoagulant yam, thiab raug collagen, uas activates lub coagulation system thiab txhawb nqa. thrombosis.
Cov ntshav khiav qeeb kuj tuaj yeem ua rau cov thrombus tsim tau yooj yim kho ntawm phab ntsa ntawm cov hlab ntsha thiab txuas ntxiv mus.

Yog li ntawd, thrombus feem ntau tshwm sim nyob rau hauv cov hlab ntsha nrog qeeb cov ntshav khiav thiab nquag mus rau eddy tam sim no (ntawm venous li qub).Aortic ntshav khiav ceev, thiab thrombus tsis tshua pom.Raws li kev txheeb cais, qhov tshwm sim ntawm venous thrombosis yog 4 zaug ntau dua li ntawm cov hlab ntsha, thiab venous thrombosis feem ntau tshwm sim hauv plawv tsis ua haujlwm, tom qab phais lossis hauv cov tsiaj muaj mob pw hauv lub zes ntev.
Yog li ntawd, nws yog ib qho tseem ceeb heev los pab cov tsiaj muaj mob uas tau pw ntev ntev thiab tom qab kev phais kom ua qee yam haujlwm tsim nyog los tiv thaiv thrombosis.
3. Kev hloov pauv hauv cov ntshav.

Feem ntau yog hais txog kev nce ntshav coagulation.Xws li kev kub hnyiab, lub cev qhuav dej, thiab lwm yam kom pom cov ntshav, kev raug mob hnyav, tom qab yug me nyuam, thiab ntshav poob qis tom qab kev ua haujlwm loj tuaj yeem ua rau cov ntshav platelets nce ntxiv, nce ntshav viscosity, thiab nce cov ntsiab lus ntawm fibrinogen, thrombin thiab lwm yam coagulation. hauv plasma nce.Cov yam ntxwv no tuaj yeem txhawb nqa thrombosis.